Does working from home increase or reduce your risk of imposter syndrome?

Thanks to the pandemic, working from home is now the norm. Instead of heading to work on cramped trains and crawling along in traffic, we’re commuting from our bedrooms to our kitchens.



a woman standing in front of a laptop: Frustrated Black businesswoman using laptop


Frustrated Black businesswoman using laptop

For some people, working from home is a welcome change. For others, though, the transition to remote working has been a challenge. Our routines have been upended, it’s hard to switch off and the days seem to blur into one, long Zoom call.

It’s normal for this kind of sudden transformation to impact the way we feel about work. In particular, it may lead to feelings of inadequacy — otherwise known as imposter syndrome — as we grapple with this new way of life.

Imposter syndrome, the fear of being outed as a fraud at any minute despite overwhelming evidence saying otherwise, is a common problem. Although your colleagues may consider you to

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TL1A and its receptor could reduce fibrosis and tissue remodeling in patients with severe lung disease

Your lungs and airways need to be stretchy, sort of like balloons. Take a big breath, and they’ll open right up.

Damaged lungs can’t open properly. Patients with asthma, idiopathic pulmonary fibrosis and systemic sclerosis suffer from fibrosis and tissue remodeling, where a build-up of tissue and immune cells, and proteins that form a glue-like substance, keep the airways from expanding. As fibrosis gets worse, taking a breath feels like blowing up a balloon filled with concrete.

In a new study, researchers at La Jolla Institute for Immunology (LJI) report that a protein called TL1A drives fibrosis in several mouse models, triggering tissue remodeling, and making it harder for lungs and airways to function normally.

“Our new study suggests that TL1A and its receptor on cells could be targets for therapeutics aimed at reducing fibrosis and tissue remodeling in patients with severe

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